相似文献/References:
[1]杨永长,传良敏,黄文芳,等.辛伐他汀抑制K562细胞MAPK1、cyclin D1、E2F1、c-myc mRNA的表达[J].陆军军医大学学报(原第三军医大学学报),2009,31(03):241.
YANG Yong-chang,CHUAN Liang-min,HUANG Wen-fang,et al.Simvastatin down-regulates mRNA expressions of MAPK1, cyclin D1, E2F1 and c-myc in K562 cells[J].J Amry Med Univ (J Third Mil Med Univ),2009,31(10):241.
[2]许晓风,李爽,李昊淼.K562细胞对树突状细胞线粒体功能和能量代谢状态的影响[J].陆军军医大学学报(原第三军医大学学报),2015,37(18):1854.
Xu Xiaofeng,Li Shuang,Li Haomiao.K562 cells impair mitochondrial function and energy metabolism of dendritic cells in Transwell coculture system[J].J Amry Med Univ (J Third Mil Med Univ),2015,37(10):1854.
[3]舒燕,齐杰玉,王灿蔚,等.MicroRNA-193b对K562细胞bcr/abl表达的影响[J].陆军军医大学学报(原第三军医大学学报),2015,37(19 ):1955.
Shu Yan,Qi Jieyu,Wang Canwei,et al.MicroRNA-193b down-regulates bcr/abl in K562 cells[J].J Amry Med Univ (J Third Mil Med Univ),2015,37(10):1955.
[4]周定安,黄文芳,刘文,等.P53途径在辛伐他汀诱导K562细胞凋亡中的作用[J].陆军军医大学学报(原第三军医大学学报),2007,29(22):2160.
ZHOU Ding-an,HUANG Wen-fang,LIU Wen,et al.Role of P53 pathway in simvastatin-induced apoptosis of K562 cells in vitro[J].J Amry Med Univ (J Third Mil Med Univ),2007,29(10):2160.
[5]周定安,黄文芳,张明川,等.辛伐他汀对K562细胞PI3K-AKT信号通路分子变化影响的体内外比较[J].陆军军医大学学报(原第三军医大学学报),2007,29(21):2049.
ZHOU Ding-an,HUANG Wen-fang,ZHANG Ming-chuan,et al.Molecular changes of PI3K-AKT signal pathway in K562 cells treated with simvastatin in vitro and in vivo[J].J Amry Med Univ (J Third Mil Med Univ),2007,29(10):2049.
[6]何鹏,骆展鹏,张伶,等.RNAi重组体抑制白血病K562细胞系中NPM1基因的表达[J].陆军军医大学学报(原第三军医大学学报),2008,30(10):938.
HE Peng,LUO Zhan-peng,ZHANG Ling,et al.RNA interference silencing nucleophosmin 1 inhibits NPM1 gene expression in K562 cells[J].J Amry Med Univ (J Third Mil Med Univ),2008,30(10):938.
[7]林雪梅,田雪莲,杨慧,等.青蒿琥酯对K562细胞和脐血CD34+造血干/祖细胞的选择性作用[J].陆军军医大学学报(原第三军医大学学报),2007,29(20):1939.
LIN Xue-mei,TIAN Xue-lian,YANG Hui,et al.Selective effect of artesunate on K562 cell and CD34+ hematopoietic stem/progenitor cell from umbilical cord blood[J].J Amry Med Univ (J Third Mil Med Univ),2007,29(10):1939.
[8]邓刚,司维柯,潘静,等.Wnt5a基因在淋巴细胞系恶性肿瘤中的表达分析[J].陆军军医大学学报(原第三军医大学学报),2008,30(19):1832.
DENG Gang,SI Wei-ke,PAN Jing,et al.Expressions of Wnt5a gene in lymphocytic malignant tumor[J].J Amry Med Univ (J Third Mil Med Univ),2008,30(10):1832.
[9]罗颖,温贤浩,徐酉华.K562细胞survivin基因表达对淋巴细胞增殖和功能的影响[J].陆军军医大学学报(原第三军医大学学报),2008,30(23):2215.
LUO Ying,WEN Xian-hao,XU You-hua.Effect of enhanced survivin expression on lymphocyte proliferation and function in K562 cells[J].J Amry Med Univ (J Third Mil Med Univ),2008,30(10):2215.
[10]史梅,冯文莉,王小中,等.靶向阻断STAT5对白血病K562细胞周期的影响[J].陆军军医大学学报(原第三军医大学学报),2005,27(21):2118.
[11]袁媛,李招权,司维柯,等.Wnt5a与髓细胞白血病发生关系的研究[J].陆军军医大学学报(原第三军医大学学报),2007,29(21):2056.
YUAN Yuan,LI Zhao-quan,SI Wei-ke,et al.Correlation between myeloid leukemia development and Wnt5a expression[J].J Amry Med Univ (J Third Mil Med Univ),2007,29(10):2056.
[12]牛长春,司维柯,李军,等.稳定过表达Wnt5a对K562细胞生物学性状的影响[J].陆军军医大学学报(原第三军医大学学报),2009,31(22):2199.
NIU Chang-chun,SI Wei-ke,LI Jun,et al.Effects of stable Wnt5a overexpression on biological characters of K562 cells[J].J Amry Med Univ (J Third Mil Med Univ),2009,31(10):2199.